Preliminary studies reveal conolidine might inhibit distinct ion channels, decreasing neuronal excitability and limiting pain alerts. This mechanism is especially relevant in neuropathic ache, where abnormal signaling brings about persistent discomfort. In addition, conolidine appears to influence G protein-coupled receptor (GPCR) pathways integral to pain notion. Computational dockin... https://ashleighn738mza4.wiki-cms.com/user